Traumatic Brain Injury In Sports

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Traumatic Brain Injury In Sports



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Traumatic brain injury: pathology review

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CT or MRI detect any bleeding in the skull. The Glasgow Coma Scale classifies the severity of brain injury, with a score of 15 as normal and progressively lower scores indicating greater neurologic injury to the brain. After testing is completed, doctors will make an estimate on the extent of the injury and possible recovery time. If cranial bleeding and swelling is minor, a short hospital stay up to a week is needed with close observation.

If bleeding is severe, the player may be treated as a patient with a severe head injury with surgery as the main option. This process requires the patient to be admitted into an intinsive care unity with close monitoring of blood levels and brain activity. The effects of a cerebral contusion depend on the cause of the injury and what part of the brain was most affected.

Outcomes vary from minor injuries that require short recovery times to severe injuries that can lead to death. Most short-term effects match that of a mild head injury while long-term effects can be much more serious. Most long-term injuries require surgery, rehabilitation, and close monitoring. In small cases, cerebral contusions can lead to death about 15 per , people. If a cerebral contusion leads to a coma, recovery can be very long and rehabilitation extensive. If the coma is long, the probability of dying or permanent neurological damage is very possible. Rules exist within each sport to help prevent cerebral contusions and traumatic brain injuries.

However, individual athletes are the best prevention against their own injuries. In a game, athletes notice when they have the symptoms of a cerebral contusion and should take themselves out of the game. It may be hard for medical personnel or coaches to notice when a player has a traumatic brain injury, so it is in the player's best interest to be removed from play. With the high percent of injuries being traumatic, extensive design improvements have been made to helmets. These improvements reduce the risk of cerebral contusions by providing more padding around the skull and a chin strap that keeps the helmet snug.

A syndrome affecting boxers that is caused by cumulative cerebral injuries and is characterized by impaired cognitive processes as thinking and remembering , Parkinsonism , impaired and often slurred speech, and slow poorly coordinated movements especially of the legs. Dementia Pugilistica DP is typically associated with the sport of boxing; although symptoms of DP may appear immediately after a single traumatic brain injury, they are typically described following the cessation of exposure to chronic brain injury.

Some of the subjective symptoms experienced after a Knockout are headaches, tinnitus, forgetfulness, impaired hearing, dizziness, nausea and impaired gait. Approximately ten percent of these active boxers reported constantly suffering from forgetfulness, headaches and other symptoms. Dementia pugilistica is difficult to diagnose until the later stages of a boxer's life. Symptoms are not apparent until boxer's are years into retirement. The septa end up separated and torn apart while the ventricles become enlarged. Segmented inversion recovery ratio imaging technique is based on the ratio of a white matter suppressed image and gray matter suppressed image.

Multiple studies have concluded that there is neurological evidence of damage to pyramidal, extrapyramidal and cerebellar systems with associated psychosis, memory loss or dementia, personality change and social instability. A hematoma is a localized collection of blood that gathers outside the blood vessels in an area it does not belong. When a direct blow to the head occurs, there is bruising to the brain and damage to the internal tissue and blood vessels.

Additionally, the jarring of the brain against the skull causes hematomas. Council on scientific affairs. An extradural hematoma is a TBI where blood collects between the inside of the skull and the dura, the thick outer covering of the brain. Blood collects on the outermost layer of the brain and creates an intracranial pressure. Generally, symptoms for hematomas are confused speech, difficulty with balance or walking, headaches, lethargy or confusion, nausea or vomiting, numbness, seizures, slurred speech, visual disturbances, and weakness. Pupils are often dilated or unequal. Additionally, hemiparesis, seizure activity, and vomiting, may be apparent. This may last for several hours while the brain function deteriorates. If untreated epidural hematoma causes increased blood pressure, shortness of breath, damage to brain function and may result in death.

Subdural and epidural hematomas are serious conditions and should be immediately diagnosed and treated by a physician. Hematomas may not show the full extent of the problem initially after the head injury, but it may be revealed after comprehensive medical evaluation and diagnostic test. The CT scan reveals evidence of blood within the skull, fractures, and signs of compression on the brain from the hematoma.

The MRI is a more thorough evaluation of injuries to the brain tissue. Yet, an MRI cannot take place if the injured victim is in a confused state. Small hematomas may be monitored closely to ensure the hematoma is not enlarging and resolved properly. Surgery reduces the pressure within the brain and stops the bleeding. The most crutial aspect for recovery in patients with severe hematomas is rapid diagnosis and appropriate treatment. Conditions which are also monitored after surgery are seizures, clot accumulation, and infection.

If complications do occur, sometime the hematoma needs to be re-drained. This is a standardized pupil response assessment of the neurologic status of the patient. GCS helps assess many different types of head injuries and predicts how a patient will recover following a hematoma. Factors such as elevated intracranial pressure, increased patients age, and abnormal GCS results lead to a poor prognosis. If an athlete is approved to return, he or she is required to complete asymptomatic at rest and with exertion. The athlete also has to clear a CT scan indicating the hematoma has entirely resolved. Lastly, the athlete needs to be slowly brought back into the sport with close monitoring to be sure the symptoms do not recur.

Preemptive measures include using safety equipment to reduce your risk of a head injury. Equipment examples are hard hats, bicycle or motorcycle helmets, and seat belts. To reduce the risk of hematomas, factors to avoid are taking anticoagulant medication blood thinners, such as aspirin , long-term abuse of alcohol, repeated falls, and reoccurring head injury. From Wikipedia, the free encyclopedia. See also: Traumatic brain injury. This article is an orphan , as no other articles link to it. Please introduce links to this page from related articles ; try the Find link tool for suggestions. January Retrieved In Herrera, Joseph E. Essential Sports Medicine. ISBN The Journal of the American Medical Association.

PMID Retrieved 1 April Sport-Related Concussion in Children and Adolescents. Sports Health: A Multidisciplinary Approach. PMC Concussion in the Adolescent Athlete. Clinics in Sports Medicine. NFL Evolution. Canadian Medical Association Journal. Clinical Sports Medicine. The Canadian Press. Merck Manuals. MD Guidelines. Am J Public Health.

NYP Health. Rolling Meadows, Illinois: Thieme. Gould, Douglas. Boxing: Medical Aspects. Academic Press. American Academy of Neurology. Retrieved 2 April Ondo March And those effects are trickling down into younger generations of sports participants. A new study published online July 29, , in the journal Brain Injury found sports and recreation to be the leading cause of nonfatal TBI in adolescents ages 5 to They wanted to determine which consumer products home furnishings and fixtures, like beds and which sports caused the most TBIs in people younger than 20 years old.

Of more than four million nonfatal TBIs seen in emergency departments, home furnishings and fixtures accounted for the highest rate of TBIs in infants less than 1 year old and children ages 1 to 4. Sports and recreation like football contributed to Football was the fifth leading cause of TBI for 5- to 9-year-olds. According to the UPMC Sports Medicine Concussion Program , around 20 percent of high school athletes who play contact sports, including soccer, sustain concussions each year. The effect these injuries have on kids largely depends on severity and early intervention.

Still, other studies, such as one published in August in the journal Brain Science , estimated that as many as 90 percent of TBI cases that pass through U. Some research shows an association between TBIs and a higher risk of developing Alzheimer's disease and dementia , and an increased risk of suicide , although more research is needed to draw any conclusions.

Park says getting an injured player out of the game or practice immediately is crucial to minimizing the impact of a brain injury. So is keeping an athlete on the sidelines until she or he is fully healed. The extent of TBI education has come a long way in recent years.